Hyperammonemic encephalopathy associated with multiple myeloma
نویسندگان
چکیده
منابع مشابه
Hyperammonemic encephalopathy in multiple myeloma.
We report two cases of hyperammonemic encephalopathy in patients with multiple myeloma. This rare complication, whose pathophysiology remains unknown, is associated with disease progression and so with a very bad prognosis. We believe that this complication should be included in the differential diagnosis of encephalopathy occurring in multiple myeloma.
متن کاملValproate-associated hyperammonemic encephalopathy.
The use of valproic acid (VPA) (also known as Depakote, Depakene, and others) frequently results in elevated plasma ammonia. In some people, hyperammonemia may be clinically significant, resulting in hyperammonemic encephalopathy, which may be severe. Valproic acid-induced hyperammonemic encephalopathy may occur in people with normal liver function, despite normal doses and serum levels of VPA....
متن کاملValproate-Induced Hyperammonemic Encephalopathy
Valproate is the best choice drug for a variety of medical conditions. As with any other drug, it has adverse effects, and it is important to emphasize the possibility of those adverse effects to prevent complications. We present the case history of a 44-year-old male with valproate-induced hyperammonemic encephalopathy, despite having normal liver function tests. This case includes a detailed ...
متن کاملNon-Hyperammonemic valproate encephalopathy
A 21-year-old male known case of primary hypothyroidism, Seizure disorder sequelae of an old trauma receiving sodium valproate, clobazam and phenobarbitone for control of Generalized tonic clonic seizures reported to neurology OPD with history of altered sensorium and gait unsteadiness for 1 week with history of hike in valproate dose 2 weeks before. On examination he was drowsy. Neurological e...
متن کامل[Valproate-induced hyperammonemic encephalopathy].
Encefalopatia hiperamonemica inducida por acido valproico.
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ژورنال
عنوان ژورنال: Neurología (English Edition)
سال: 2017
ISSN: 2173-5808
DOI: 10.1016/j.nrleng.2015.08.010